ObjectiveTo establish a ideal experimental rat model of acute necrosis pancreatitis (ANP).
目的建立一种理想的急性坏死性胰腺炎(ANP)大鼠模型。
Conclusion OXPAPC blocked LPS signal pathway, thus decreased the severity of experimental ANP in rat.
结论OXPAPC可阻断内毒素信号通路,从而显著降低实验性anp的严重程度。
Methods The rat model of ANP was established by retrograde bile-pancreatic duct injection of 5% sodium taurocholate.
方法逆行胆胰管注射5 %牛磺胆酸钠溶液制成ANP模型。
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